學術著作
期刊論文 (since 1992):
1. Kuo, C.-C., and Hess, P. (1992) A functional view of the entrances of L-type Ca2+ channels: estimates of the size and surface potential at the pore mouths. Neuron 9:515-526
2. Kuo, C.-C., and Hess, P. (1993) Ion permeation through the L-type Ca2+ channel in rat phaeochromocytoma cells: two sets of ion binding sites in the pore. Journal of Physiology 466:629-655
3. Kuo, C.-C., and Hess, P. (1993) Characterization of the high-affinity Ca2+ binding sites in the L-type Ca2+ channel pore in rat phaeochromocytoma cells. Journal of Physiology 466:657-682
4. Kuo, C.-C., and Hess, P. (1993) Block of the L-type Ca2+ channel pore by external and internal Mg2+ in rat phaeochromocytoma cells. Journal of Physiology 466:683-706
5. Kuo, C.-C., and Bean, B.P. (1993) G-protein modulation of ion permeation through N-type calcium channels. Nature 365:258-262
6. Kuo, C.-C., and Bean, B.P. (1994) Na+ channels must deactivate to recover from inactivation. Neuron 12:819-829
7. Kuo, C.-C., and Bean, B.P. (1994) Slow binding of phenytoin to inactivated sodium channels in rat hippocampal neurons. Molecular Pharmacology 46:716-725
8. Geula, C., Mesulam, M.-M., Kuo, C.-C., and Tokuno, H. (1995) Postnatal development of cortical acetylcholinesterase-rich neurons in the rat brain: permanent and transient patterns. Experimental Neurology 134:157-178
9. Kuo, C.-C. (1997) Deactivation retards recovery from inactivation in Shaker K+ channels. Journal of Neuroscience 17:3436-3444
10. Kuo, C.-C., Chen, R.-S., Lu, L., and Chen, R.-C. (1997) Carbamazepine inhibition of neuronal Na+ currents: quantitative distinction from phenytoin and possible therapeutic implications. Molecular Pharmacology 51:1077-1083
11. Kuo, C.-C., and Lu, L. (1997) Characterization of lamotrigine inhibition of Na+ channels in rat hippocampal neurons. British Journal of Pharmacology 121:1231-1238
12. Kuo, C.-C. (1998) A common anticonvulsant binding site for phenytoin, carbamazepine, and lamotrigine in neuronal Na+ channels. Molecular Pharmacology 54:712-721
13. Kuo, C.-C. (1998) Imipramine Inhibition of transient K+ current: an open-channel blocker preventing fast inactivation. Biophysical Journal 12:2845-2857
14. Shieh, R.-C., Chang, J.-C., and Kuo, C.-C. (1999) K+ binding sites and interactions between permeating K+ ions at the external pore mouth of an inward rectifier K+ channel (Kir2.1). Journal of Biological Chemistry 274:17424-17430
15. Kuo, C.-C., and Chen F.-P. (1999) Zn+ modulation of neuronal transient K+ current: fast and selective binding to the deactivated channels. Biophysical Journal 77:2552-2562
16. Kuo, C.-C., Huang, R.-C., and Lou, B.-S. (2000) Inhibition of Na+ current by diphenhydramine and other diphenyl compounds: molecular determinants of selective binding to the inactivated channels. Molecular Pharmacology 57:135-143
17. Kuo, C.-C., and Liao, S.-Y. (2000) Facilitation of recovery from inactivation by external Na+ and location of the activation gate in neuronal Na+ channels. Journal of Neuroscience 20:5639-5646
18. Kuo, C.-C., and Yang, S. (2001) Recovery from inactivation of T-type Ca2+ channels in rat thalamic neurons. Journal of Neuroscience 21:1884-1892
19.Yang, Y.-C., and Kuo, C.-C. (2002) Inhibition of Na+ current by imipramine and related compounds: different binding kinetics as an inactivation stabilizer and as an open channel blocker. molecular Pharmacology 62:1228-1237
20.Kuo, C.-C., Lin, T.-J., and Hsieh, C.-P. (2002) Effect of Na+ flow on cd2+ block of tetrodotoxin-resistant Na+ channels. Journal of General Physiology 120:159-172
21.Yang, Y.-C, and Kuo, C.-C. (2003) The position of the fourth segment of domain 4 determines status of the inactivation fate in Na+ channels. Journal of Neuroscience 23: 4922-4930
22.Kuo, C.-C., Lin, B.-J., Chang, H.-R., and Hsieh, C.-P.(2004) Use-dependent inhibition of the N-Methy-D-asparate currents by felbamate: a gating modifier with selective binding to the desensitized channels. molecular Pharmacology 65: 370-380.
23.Kuo, C.-C., Chen, W.-Y., and Yang, Y.-C. Yang. (2004) Block of tetrodotoxin-resistant Na+ channel pore by multivalent cations: gating modification and Na+ flow dependence. Journal of General Physiology 124: 27-42.
24.Yang, Y. -C., and Kuo, C. -C. (2005) An inactivation stabilizer of the Na+ channel acts as an opportunistic pore blocker modulated by external Na+. Journal of General Physiology 125: 465-481.